These groups were subsequently divided into 3 subgroups (n=6/subgroup): Early Euthanasia (EE), Mid Euthanasia (ME) and Late Euthanasia (LE), killed at the 6th, 15th and 21st days respectively. While chromosome aberations, micronuclei and sperm head anomaly were generally reduced in the drug-fed series, the mitotic index showed an apparent increase. Mercurius solubilis is commonly used for conditions associated with infections of the throat, mouth, ear and urinary tract.

A critical analysis of the data revealed that in the post-fed series the degree of suppression was visibly greater than in the pre-fed series in most of the parameters studied. It may accumulate in the body, with the target organs being the Kidneys and the Brain; eventually and potentially causing severe illness, decay, disintegration and death. Efficacy of potentized homeopathic drug (Arsenicum Album-30) in reducing genotoxic effects produced by arsenic trioxide in mice: III Enzymatic changes and recovery of tissue damage in liver.

This drug in its crude form has the power to change healthy cells into inflamed and necrotic wrecks, decompose the blood, and produce profound anemia.

Methyl mercury-induced chromosomal damages have been reported in lymphocyte culture ( 9) of 23 persons who ingested fish exposed to mercury-contaminated water. This medicine works well to treat ear complaints including earache, ear discharges (otorrhoea) and ear infections. G–J) Bone marrow smears showing micronucleated erythrocytes: ( G and H) polychromatic and ( I and J) normochromatic erythrocytes. Quantitative and qualitative changes were observed in the perialveolar microbiota when the groups were compared.In the present study, repair of chromosome damage has been noted in a steadily enhanced rate in the homeopathic drug-fed mice as compared to that in the mercuric chloride-treated but drug-unfed mice. e. pre-feeding, post-feeding and combined pre- and post-feeding, for their possible efficacy in ameliorating mercuric chloride-induced genotoxicity in mice. Generally, 3–4-months-old healthy adult mice of both sexes weighing 22–28 g were selected for the study.

e. assays of chromosome aberrations, micronuclei, mitotic index and sperm head anomaly were adopted in the present study to determine the genotoxic effects. In the present study, more divisional activity was necessary to replenish the cells that might have been lost owing to the clastogenic and mitotoxic effects of mercuric chloride.That carries only the energy and imprint of the Mercury characteristics, and leaves no physical substance.